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PRELIMINARY RESULTS OF A PHASE I/II STUDY OF AZACITIDINE, VENETOCLAX AND PEVONEDISTAT IN PATIENTS WITH SECONDARY ACUTE MYELOID LEUKEMIA WHO ARE UNFIT FOR INTENSIVE CHEMOTHERAPY

LPA3-mediated lysophosphatidic acid signaling promotes postnatal heart regeneration in mice

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Endothelial cell dysfunction is an initial step in atherosclerotic lesion formation and is more likely to occur at arterial curves and branches that are subjected to low shear stress and disturbed blood flow (atherosclerosis prone areas) (7,8)

Reduction of skin innervation is associated with a severe fibromyalgia phenotype

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