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However, in states of chronic illness, NF-B becomes chronically activated, triggering the continuous production of pro-inflammatory cytokines like Interleukin-6 (IL-6) and Tumor Necrosis Factor-alpha (TNF-), as well as inflammatory enzymes like Cyclooxygenase-2 (COX-2) and Lipoxygenase (LOX)

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1919 and 2012.2 GBM cell models with comparable proliferation rates, were obtained from an intermediate proneural (PN) GBM tumor and a PN GBM tumor, respectively, as previously described [42]