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Given the limitations and side effects of existing treatments for neuropsychiatric disorders, antioxidant therapy presents a promising, safer alternative

GSH selenium-based enzyme family glutathione peroxidase Glutathione reductase GR GSH to GSSG ratio can be used as a readout of tissue redox state (Zitka et al.)

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Within neurons, excess Fe + undergoes Fenton or HaberWeiss reactions, generating hydroxyl radicals that attack DNA, proteins, and lipid membranes, thereby inducing ROS and lipid peroxidation (183)

Its recognition and quantification could transform a vague idiopathic diagnosis into a defined, mechanistically driven endophenotype with direct therapeutic implications