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Depletion of total sulfide levels (a representation of H 2 S concentration [325]) during infection was observed here

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Changes in a significant number of other genes were thought to be PD-causative and were discovered by linkage analysis or a candidate gene approach, in addition to the genes responsible for the six monogenic types of PD

During this process, a small fraction of proliferating myoblasts return back to quiescence to replenish the stem cell pool 9

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Oxidation of lipid rafts and synaptic vesicle membranes alters voltage-gated channels, N-methyl-D-aspartate (NMDA), -amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptors (AMPA) and kainate receptor (KAR) localization consequently impairing neuronal function and making neurons vulnerable to excitotoxicity leading to neurodegeneration (Mattson, 1998