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Tumour-induced glucose restriction in the TME reduces NK cell glycolysis and disrupts normal NK cell effector functions, such as interferon-gamma (IFN-) production and target cell damage, ultimately suppressing antitumour capacity ( 3.3.4 Dendritic cells As is with macrophages, dendritic cells (DCs) can rapidly respond to invading pathogens and they constitute the bodys early defense barrier

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The combination AZT plus indinavir induced an increase in ROS production, disruption in membrane mitochondrial potential, reduction in intracellular GSH levels, augment permeability of endothelial layer, leading cell death [49]

The metabolic demands placed upon a cell from the stresses associated with proliferation caused by oncogenic transformation produce unique needs that must be met through extracellular sources of cysteine and de novo cysteine generation [5,21,22]