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Verhey, T

These macrophage-derived metabolic signals synergistically increase lipid peroxidation through the ACSL4-dependent synthesis of PUFA-phospholipids, pushing cancer cells beyond their antioxidant capacity and into a state of ferroptosis

increased levels of proinflammatory mediators and inflammatory potential

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In addition, oxidative stress results in alterations in the local immune response, increasing the risk of infections and exacerbations, which, in turn, may accelerate lung function decline

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