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Their beneficial mechanisms can include the attenuation of neuroinflammation [e.g., valproate via histone deacetylase (HDAC) inhibition in microglia, downregulating interleukin-1 beta (IL-1) and inducible nitric oxide synthase (iNOS)] or the mitigation of glutamatergic excitotoxicity coupled with enhancement of neurotrophic factor expression (e.g., lamotrigine), thereby conferring antidepressant-like potential (Mula and Sander, 2007

Tm nhn ca chng ti khng ch l vic to ra nhng sn phm lm p xut sc, m cn l v vic xy dng mt cng ng ph n mnh m, t tin v quyn lc

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DiNardo C, Olin R, Ishizawa J, Sumi H, Xie J, Kato K, Kumar P, Andreeff M