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Mitochondrial dysfunction itself can be categorized into two types: the first type represents a mutation of a gene which is associated with the ATP-generating pathway, while the second one is related to various abnormalities such as genetic, biochemic or metabolic, that may weaken the ability of the mitochondria to produce ATP (Haas et al., 2007)

developed inhalable dexamethasoneloaded PLGAlipid nanoparticles with surface modification of FcRn peptide, which facilitates transepithelial transport of nanoparticles and subsequent entry into inflammatory cells [58]

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It safeguards the CNS from harmful toxins, pathogens, and foreign substances in the blood, while simultaneously supplying vital nutrients to brain tissue and eliminating waste products[4]