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This decline coincides with reduced regenerative capacity, prompting researchers to investigate its potential for supporting skin health

Subsequent research indicated that the enhancement of MET by activating UPF1 (a versatile RNA and DNA helicase) caused phosphorylation, which, in turn, controlled the 3'-UTR length of STING, leading to a decrease in STING expression in cancer cells [123]

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Further experiments revealed that the p62-Keap1-NFE2L2 pathway inhibits ferroptosis