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Description
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Coordinates and velocities of the equilibrated systems were used for 50 ns production simulations performed under the conditions described above
show that amlodipine may increase flow-mediated dilation, NO, and eNOS activity in human umbilical vein endothelial cells through decreased PKC phosphorylation, and reduced PKC-dependent threonine 495 phosphorylation on eNOS (144)
doi: 10.1016/S0899-9007(03)00024-8
