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PPARs are promising therapeutic targets for neurodegenerative diseases, including PD 63 and LID 64 , due to their neuroprotective effects against oxidative damage and regulatory role in lipid metabolism, along with development, tissue differentiation, inflammation and mitochondrial function 63

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The results demonstrated that BV2-metformin-loaded PLGA treatment reduced TNF- and GFAP expression, confirming suppression of neuroinflammation, while p62 levels were increased, indicating enhanced autophagy