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however, the success of the HSC gene therapy shows that only a partial correction of HSC progeny is necessary to halt cerebral disease [37, 95]

Coptisine protects against hyperuricemic nephropathy through alleviating inflammation, oxidative stress and mitochondrial apoptosis via PI3K/Akt signaling pathway

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Therefore, timely intervention and proper treatment are crucial in addressing B12 deficiencies and maintaining overall bodily health

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Additionally, it was found that MeJA treatment promoted VOC accumulation and regulated the antioxidant system, including the ascorbate-glutathione (AsA-GSH) cycle [1]