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Intriguingly, however, Cu (II)-atsm does not improve astrocyte dysfunction by increasing SOD1 activity but by decreasing mitochondrial respiration, which is increased in these cells, and by increasing glycolysis (Dennys et al., 2023)

These results highlight tissue-specific mitochondrial reprogramming to support liver energetic demands and antioxidant synthesis, rather than global mitochondrial dysfunction, as a metabolic consequence of sepsis

An evaluation of the stability and pharmacokinetics of R-Lipoic acid and R-Dihydrolipoic acid dosage forms in human plasma from healthy subjects

Catecholamine, indolealkylamine and calcium levels of human pineal glands in various clinical conditions

There has also been an increased incidence in Southern Europe, the Middle East, Southeast Asia, and Oceania

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