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doi: 10.20524/aog.2021.0571

Given the multi-factorial nature of secondary injury, many different therapeutic approaches have been investigated in an attempt to mitigate the post-acute neuronal damage caused by TBI including antioxidants, neurorestorative therapies, neuroprotective pharmacological agents, and drugs that modulate neuroinflammation (Hall et al., 2010

In addition, MTHFD2 deficiency also promotes Treg cell differentiation (75)

Their effects are mediated via NK1 receptors (mainly for SubP) and NK2 receptors (activated by NKA)

Gelatin acts like a soft cushion between bones that helps them move smoothly without friction