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This enzyme is required to maintain a cellular redox state by sustaining a balance between reduced Glutathione and AsA pool by regenerating the reduced form of GSH [194]

Post-transcriptional mechanisms that regulate GPx-1 activity may prove to be crucial to maintaining cellular redox balance

Similar content being viewed by others Introduction The kidneys sensitivity to xenobiotics and role in maintaining bodily homeostasis are crucial 1

FA significantly suppressed Ang II-induced expression of aging biomarkers (p16, p21, and VCAM-1), reduced the increase of -galactosidase, and promoted HAECs proliferation (Fig

& Desnues, C

Br J Pharmacol 174, 13251348.CrossRefGoogle ScholarPubMed Hansen, M, Rubinsztein, DC & Walker, DW (2018) Autophagy as a promoter of longevity: insights from model organisms