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By comparing AOD 9604 with GHRH analogues and full-length growth hormone, researchers can better define pathway specificity, receptor involvement, and downstream metabolic outcomes in controlled laboratory models

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J Cell Biol

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In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis

Methylcobalamin and adenosylcobalamin represent the two active forms that participate directly in cellular metabolism