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This works quite well in lower demand patients, especially after distal radius fractures that have healed in abnormal alignment, in patients with this bone, where realigning the radius might not work

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Although iron-dependent prolyl hydroxylases (PHDs) regulate both isoforms and iron chelation can stabilize HIF-1 and HIF-2 in vitro (74), these observations collectively suggest that the gut may implement additional, tissue-specific mechanisms that bias HIF isoform activity and downstream iron handling