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10.1016/j.ebiom.2018.08.047 92 NilsenG

We integrate recent discoveries, highlight, and critically discuss the underlying biochemical and molecular pathways involved

Cell Mol Life Sci 74(21):39133925

Shelise Edwards, MD, FACEP At enTrust Immediate Care, we believe exceptional healthcare means more than treating symptoms it means listening, caring, and delivering expert medical attention when patients need it most

Lu, W., Wang, L., Chen, L., Hui, S

Consequence of Nrf2 upregulation during tumorigenesis The above data suggest that although the upregulation of Nrf2 in cancer seems paradoxical, it is not an inconsequential observation or artifactthe mutations seen are specifically targeted to activate and cause accumulation of Nrf2, either by increasing stability of the transcription factor (by inhibiting its primary negative regulator Keap1 or suppressing repression by -TrCP) or by increasing transcription of NFE2L2