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furthermore, the in vitro migration and rate of spreading of tendon fibroblasts increased in a dose-dependent manner, which was attributed to the activation of the FAK-paxillin pathway

and mass spectrometry confirming both molecular weights (BPC-157: 1419.53 Da

Again, these findings are relevant solely to biochemical and molecular research and are not indicative of protective effects in living organisms

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1,12 [corrected] Clinical evaluation seems to show an inverse relationship between the severity of megaloblastic anemia and the degree of neurologic impairment

Front Neuroendocrinol 2019;54:100772