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332,333 In CAFs coupled with prostate carcinoma cells models, contact between tumors and the stroma-activated CAFs through stabilization of HIF-1 that rely on sirtuin 3 or succinate, initiating mitochondrial oxidative stress, promoting mitophagy, upregulating expression of GLUT1 and glycolytic enzymes, and facilitating lactate biosynthesis

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Paradoxically, this same mechanism may underlie its potential pro-cancer effects: by boosting GSH production, cancer cells can better protect themselves from oxidative damage caused by ROS (Ramachandran and Jaeschke, 2021)

10.1016/s8756-3282(98)00180-x [DOI] [PubMed] [Google Scholar] Seiwerth S., Brcic L., Vuletic L

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