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Ferroptosis is strikingly driven by worsening redox imbalance, which generates and maintains high levels of hydroperoxy-phospholipids (7, 24)

This exosome-based signaling cascade transforms ferroptosis from a localized neuronal death event into a spatially expanding pathological phenomenon within neural networks

Contributor Information Ying Zhang, Email: [email protected]

At 3.5 mM cysteine, ~9% of the overall sample composition comprised [4Fe4S] 2+ clusters (grey dashed line) 43,44,47 , with an isomer shift of 0.35 mm/s and E q of 1.01 mm/s, while the remaining 26.3% was attributed to ferrous iron with an expanded (either 5 or 6) coordination 44,48 (orange dashed line, of 1.02 mm/s and E q of 2.97 mm/s)

Impaired renal function and dysbiosis of gut microbiota contribute to increased trimethylamine-N-oxide in chronic kidney disease patients

88 , 3638 (2016) M.D