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In endothelial cell models, BPC-157 significantly enhances ERK1/2 phosphorylation in a dose-dependent manner, leading to increased cellular proliferation, migration, and vascular tube formationprocesses central to tissue regeneration and angiogenesis [26, 55]

When repair falls short, tissue integrity is still at least initially maintained by independent stress-response mechanisms as apoptosis and cellular senescence(de Keizer, 2017)

115 ScottS
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