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In terms of pathophysiological mechanisms of PD, the A2A receptor attracted attention due to its high striatopallidal expression, specifically in GABAergic and glutamatergic striatopallidal medium spiny neurons (Fig

5-Amino-1MQ is an orally bioavailable, cell-permeable small molecule that potently inhibits NNMT (nicotinamide N-methyltransferase), the enzyme responsible for methylating nicotinamide to 1-methylnicotinamide (1-MNA)

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Louvet A, Labreuche J, Artru F, Bouthors A, Rolland B, Saffers P, et al

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[DOI] [PubMed] [Google Scholar] 72.Balenovic D., Bencic M.L., Udovicic M., Simonji K., Hanzevacki J.S., Barisic I., Kranjcevic S., Prkacin I., Coric V., Brcic L., et al