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While autophagy suppresses apoptosis and pyroptosis by degrading Caspase-8 [162], excessive autophagy (autophagic cell death) releases unprocessed autophagosomes containing free fatty acids and ROS (O 2 - , H 2 O 2 ), activating RIPK1-dependent necroptosis [163]

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[] Reduced oxidative stress, neuroprotection of cognitive networks and increased glutamatergic regulation of the NMDA receptor system are some ways that NAC could enhance cognitive abilities amongst patients with schizophrenia