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The fact that wild type SnCE1 shows less mitochondrial fragmentation compared to SnCE1 Y212A, although both are monomeric and active deSUMOylases, might be because the proteolytic processing is only triggered after SnCE1 deacetylation for wild type SnCE1 while SnCE1 Y212A is not acetylated when it is produced and autoproteolytic processing can start immediately following translocation into the host cell as supported by the more pronounced processing observed for SnCE1 Y212A in HEK-293T cells (Fig

Franoise Barr-Sinoussi, Honorary President of the Virology Department at the Institut Pasteur and of the Institut Pasteur International Network, and also President of the French AIDS charity Sidaction since November 2017, told us the story of this discovery when we spoke to her over the summer

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Antioxidant treatment is known to reduce the progression of HT and even prevent its development (da Silva et al

* Correspondence: Hongzhi Yu, [email protected]

Investigators often reference primary literature on platforms such as PubMed when designing protocols, and consult repositories at the NCBI and the FDA for context on analytical and regulatory standards