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without screening, the disease may not be identified and treated

Child Fetal Neonat

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de Villiers WJ, van der Westhuyzen DR, Coetzee GA, Henderson HE, Marais AD

Pain Processing Mechanisms Central Sensitization (via mGlu2 receptor upregulation)[4][5] Neuroinflammation (modulates glial activation)[12][13] Mitochondrial Dysfunction (enhances ATP production, -oxidation)[2][13] Oxidative Stress (direct antioxidant activity)[2][13] Systemic Inflammation limited direct evidence SUMMARY OVERVIEW CLINICAL PEARLS 1

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