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No IGF-1 elevation: AOD-9604 does not stimulate IGF-1, meaning it avoids risks such as organ/tissue overgrowth or acromegaly

Deactivation where the products of free radical fractions are repaired or eliminated, such as by antioxidants that can repair damaged cells and proteins

Mechanism of Action: Dihexa binds with high affinity to hepatocyte growth factor (HGF), activating c-Met receptors, which leads to increased synaptic connectivity and neurogenesis

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HMGB1, a typical DAMP, is an endogenous mediator of inflammation that can be released through apoptosis, ferroptosis, and necrosis (217)

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