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GHK-Cu works through copper delivery to metalloenzymes and broad gene expression modulation, affecting not only collagen synthesis but also collagen cross-linking (via lysyl oxidase), glycosaminoglycan production, anti-inflammatory pathways, and antioxidant defense systems

The expression levels of immune checkpoint molecules were compared across the risk groups and the results demonstrated that TNF superfamily member (TNFSF)18, CD160, adenosine A2a receptor, neuropilin-1, CD200 and TNFSF9 were markedly upregulated, while CD44 was markedly downregulated in the high-risk group compared with the low-risk group (Fig

Funding The authors declare that there are no sources of funding to be acknowledged

Liu Y, Pan Y, Gao W, et al

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