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In cases of resistant tumors harboring JAK mutations that impair IFN signal transduction, novel immunotherapeutic approaches that bypass this defective pathway (such as those leveraging IFN-independent cytotoxic mechanisms) or bispecific antibodies offer a critical strategy to overcome treatment resistance

pylori gGT may mediate bacterial acquisition of Cys-Gly from extracellular GSH

[DOI] [PubMed] [Google Scholar] 215.Henden T., Stokkan K.A., Reiter R.J., Nonaka K.O., Lerchl A., Jones D.J