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These alterations contribute to systemic inflammation by increasing the production of pro-inflammatory cytokines, including interleukin (IL)-1, IL-6, IL-8, IL-13, tumor necrosis factor-alpha (TNF-), and transforming growth factor-beta 1 (TGF-1) [24]

aureus in its induction of an inflammatory response within the mammary gland of mice increased the levels of IL-1, IL-6, and TNF-

The proinflammatory effects of mCRP in the alveoli may be inhibited by PC in lung surfactant, which is abundant in the terminal airway where the major lipid component is dipalmitoyl phosphatidylcholine that has a polar head group consisting of PC (214)

K: Western blot, n = 4 for each group)

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