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Moreover, the relapse-preventing effects of NAC are believed to arise chiefly from the restoration of extrasynaptic glutamate to a physiological level, thereby preferentially activating mGluR2/3 receptors [27]
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Various Possible Toxicants Involved in Thyroid Dysfunction: A Review
doi: 10.1002/hep.1840150631
FoxP3 isoforms and PD-1 expression by T regulatory cells in multiple sclerosis
