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This enhanced oxidative stress leads to cell damage resulting in various complications such as sepsis, myocardial injury and increased mortality

Hyperammonaemia was associated with a multitude of aetiologies, the most common of which were inborn errors of metabolism

Over the next decades, this has prompted several modifications to make ADT increasingly more complete and potent in inhibiting AR, which have been the subject of excellent reviews [4, 7, 8]

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Additionally, activation of the nuclear factor kappa-B (NF-B) pathway can promote cell survival by upregulating anti-apoptotic genes, thereby contributing to cisplatin resistance (59)

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