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The FDX1-mediated process culminates in profound proteotoxic stress driven by the aggregation of lipoylated mitochondrial proteins and the concomitant destabilization of Fe-S cluster proteins, which together lead to acute mitochondrial failure [3]

However this step is rate limited thus when the rate of 5-oxoproline production exceeds the rate at which it is metabolised it will accumulate

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Egr-1 mediated cardiac miR-99 family expression diverges physiological hypertrophy from pathological hypertrophy

Seleniums role in plant secondary metabolism: regulation and mechanistic insights

Ann Thorac Surg 111(4):1111-1117, 2021