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Therefore, it can have a toxic effect on -synuclein fibrillation by increasing the number of oligomeric species and protofibrils rather than full length fibrils (Kumari et al., 2022)

The peptide achieves these complex neurological stabilizations largely through intricate, bidirectional interactions with the nitric oxide (NO) system, dynamically responding to both NO-system blockade (via L-NAME administration) and NO-system over-stimulation (via L-arginine administration) to restore neurological homeostasis

In addition, the loss of neurons and the density of cholinergic neurons were also mitigated [198]

Matrix recoveries were performed in BPA spiked tissues and averaged 104 10%

Ganong, 2000)

[DOI] [PubMed] [Google Scholar] 371.St-Pierre J, Buckingham JA, Roebuck SJ, Brand MD