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Physiol Paris

Dysregulation of this pathway through altered dimerization, post-translational modifications, or impaired regulation contributes to the pathophysiology of Alzheimers disease, Parkinsons disease, Huntingtons disease, and amyotrophic lateral sclerosis, leading to protein aggregation, mitochondrial dysfunction, and neuroinflammation

Oxidative stress induces both phosphorylation and the GS-ylation of STAT1 causing the hyper-activation of its signaling in microglia cells, pointing out the importance of this transcription factor in neuroinflammation in several neurological diseases [94,95]

As noted earlier, IL-6 is often significantly upregulated in the synovial fluid and serum of OA patients

In mice, the effect of SkQ1 is also accompanied by significantly slowing down most age-related processes such as osteoporosis, sarcopenia or loss of vision [135,136]