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In vitro experiments in NIH-3T3 cells revealed that the combination treatment downregulated -SMA expression and inhibited JAK2, STAT3, and STAT1 signaling pathways, reducing fibroblast activation and migration induced by TGF-1 and IL6, thereby suppressing fibrosis progression through the JAK2-STAT3/STAT1 pathway

doi: 10.1038/sj.bjp.0705088 213 TarlatzisBKolibianakisEBontisJTousiouMLagosSMantalenakisS

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10.18632/oncotarget.5162 54 LachaierL

[DOI] [PubMed] [Google Scholar] 583.Benedetti, R

This limits the widespread application of ferroptosis-targeting therapies in clinical practice