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Description
In a mouse model of pressure-overload heart failure, the inhibitor significantly alleviated myocardial remodeling and dysfunction by inhibiting H 2 S degradation, demonstrating the feasibility of targeting the H 2 S metabolic pathway for treating heart failure
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Proc Natl Acad Sci USA 103(35):1318013185
As depicted in Figure 3, we propose that the lactate-lactylation axis drives PH pathogenesis through five key mechanisms: PASMC proliferation, immune-inflammatory responses, pulmonary fibrosis, abnormal vasoconstriction, and PASMC ferroptosis
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