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In AD, a progressive neurodegenerative disorder, disruptions in energy metabolism contribute to the gradual degeneration and apoptosis of neuronal cells in the brain, ultimately causing severe cognitive impairment

Our data indicate a potential acetyl-cystein state only occurring transiently as acetylated wild type SnCE1 is active as deSUMOylase and an accessible Cys256 is essential for catalysis

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Gene Therapy Approaches