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The Hepatitis C virus (HCV) relies on the hosts production of phosphatidylcholine for its own replication, a mechanism driven by the viral NS3/4A protease

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luckily, we achieved that goal

Thyroxine is peripherally deiodinated to form triiodothyronine which exerts a broad spectrum of stimulatory effects on cell metabolism

(2019) 52:e12570

trial (NCT03608553), which is still in progress
