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The active 15-amino-acid fragment of that protein became known as BPC-157

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The role of MET in chemotherapy resistance

By contrast, mTOR is inactive under nutrient-poor conditions: dephosphorylated ULK1 dissociates from the mTOR complex and then phosphorylates Atg13 and RB1-inducible coiled-coil (1RB1CC1/FIP200), thus triggering autophagy [26]
