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[DOI] [PubMed] [Google Scholar] 86.Sarfstein R., Pasmanik-Chor M., Yeheskel A., Edry L., Shomron N., Warman N., Wertheimer E., Maor S., Shochat L., Werner H
Authors proposed a model wherein PL enters the cell, is hydrolyzed in its active form (hPL) with subsequent formation of a hPL:GSH complex that binds the active site of GSTP1-1 causing its inhibition (Fig
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