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in the other, the primary problem is an acute event that triggers glutamatergic over-activity, excitotoxicity, neural damage and a subsequent deficit state

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[DOI] [PubMed] [Google Scholar] 130.Anisimov V

Taurine protected myocardial mitochondria injury induced by hyperhomocysteinemia in rats

Furthermore, glutamine metabolism seems to be critical for NKT cell homeostasis and mitochondrial functions

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