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Zhu X, Skoneczny D, Weidenhamer JD, Mwendwa JM, Weston PA, Gurr GM et al

These findings strongly suggest that the disease-promoting effect of eosinophils does not solely result from their ability to release IL-4 and to elicit a Th2 response, but involves additional mechanisms that support pathogen survival and chronic inflammation

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Additionally, DSS treatment decreased occludin gene expression and claudin-1 expression in immunohistochemistry (IHC) (79)

However, we consider it more likely that genetic variants that are common to BL/6J, N5-WT and N5-Tr mice vs BL/6N mice, or vice versa, contribute largely to the differences in glucose tolerance between BL/6N and N5-WT mice, as discussed earlier [13, 17]

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