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Data regarding potential OS benefit are still pending

Everyone in the family worked

Brahm, A

Understanding the pathophysiological mechanism of TGRLs metabolism and targeting LPL as a key molecule with recent therapeutic approach in the treatment of hypertriglyceridemia can be a very bright signal to optimize patients with intractable TG levels and with high residual risks for ASCVDs

[Crossref] Liang Y, Tian R, Wang J, Shan Y, Gao H, et al

CaiD was initially suggested to function as the racemase, as the caiD gene is required for racemase activity (Eichler et al
