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none of these pathway effects survived FDR correction (Fig

10.3748/wjg.v17.i12.1569 134

Several endogenous metabolites, including ubiquinol, vitamin E, and vitamin K hydroquinone, function as radical-trapping antioxidants that terminate lipid peroxyl radicals and suppress ferroptotic membrane damage 84-87

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Reactive oxygen species induce injury of the intestinal epithelium during hyperoxia

6b), thus allowing the development of FASN-positive tumor foci
