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Abstract Parkinson's disease (PD) is a major global health challenge, and the identification of highly specific and easily detectable blood markers is critical for early diagnosis and intervention in PD

doi: 10.1007/s12011-017-1085-0, 132

2020 Jul 17;7:CD006634

This metabolism-immunity crosstalk disrupts both innate and adaptive immunity: it drives the aberrant activation of macrophages and neutrophils while reprogramming T cell metabolismenhancing glycolysis in pro-inflammatory Th1/Th17 cells and impairing mitochondrial oxidative phosphorylation in Tregsultimately establishing a chronic intra-renal inflammatory microenvironment that accelerates glomerulosclerosis and tubulointerstitial fibrosis

In contrast, these disruptions were largely prevented by cotreatment with etomoxir (Figure 2A) as confirmed by two independent, blinded scorers (Figure 2B)