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Using lipid nanoparticle-encapsulated siRNA to target and silence macrophage Cxcl2 markedly reduces myocardial neutrophil infiltration, diminishes inflammation, and reduces infarct size, concurrently demonstrating that aberrant macrophage chemotactic activity is a key target exacerbating post-inflammatory myocardial ischemic injury (33)

JunD, not c-Jun, is the AP-1 transcription factor required for Ras-induced lung cancer

Our results indicate that lipoproteins bind and regulate CLCF1 activity, suggesting that variation of the ratio between the different lipoproteins in serum or other body fluids could control the neurotrophic, metabolic and immunoregulatory functions of CLCF1

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