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Mice lacking the modifier subunit of GCL, have a 60% reduction in glutathione, accompanied by abnormal cortical gamma synchrony, decreased parvalbumin interneurons (PV-IN) and behavioral phenotypes relevant to schizophrenia.(49-51)

Central to this evaluation is a rigorous assessment of pathogenic potential, including analyses of a strains ability to cause infection or produce harmful metabolites (544)

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These enzymes include heme oxygenase-1 (HO-1), which degrades heme and exhibits antioxidant and anti-inflammatory effects, NAD(P)H:quinone oxidoreductase 1 (NQO1), which aids in quinone detoxification, and glutamate-cysteine ligase modifier subunit (GCLM), which is crucial for glutathione synthesis

Wickham S, West MB, Cook PF, Hanigan MH

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