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PLoS ONE 5, e11552 (2010)

Defining subtype-specific stress-response patterns and therapeutic vulnerabilities may therefore represent an important direction for future research

Combined GSK-3 inhibitor and Nrf2 activator therapies may be an advantage, as additive protective effects against A toxicity are observed with Keap1 mutation and lithium treatment in the fly (Kerr et al., 2017) and DMF has recently been shown to prevent tau phosphorylation through inhibition of GSK-3 and neuronal damage by subsequent activation of Nrf2 through Keap1 dependent and independent mechanisms (Cuadrado et al., 2018)

Reiter, R., and Oram, J

500 Zhang et al

Muscle repair demands more deep sleep than the body is getting