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Homocysteine, a risk factor for premature vascular disease and thrombosis, induces tissue factor activity in endothelial cells

The increase of intracellular ATP levels hesitates in the closure of plasma membrane K+ channels, thus triggering -cell depolarization

This was likely because they burned more fat while preserving muscle glycogen, in addition to having lower levels of lactic acid and higher levels of creatine phosphate

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